Jianxin, W., Jiayu, X. & Yaozong, Y. Effects and mechanisms of emodin and sandostatin on pancreatic ischemia in acute haemorrhagic necrotizing pancreatitis., CJIM 4, 34–38 (1998). https://doi.org/10.1007/BF02935047
Wu Jianxin, Xu Jiayu, Yuan Yaozong. Effects and mechanisms of emodin and sandostatin on pancreatic ischemia in acute haemorrhagic necrotizing pancreatitis. [J]. Chinese Journal of Integrative Medicine 4(1):34-38(1998)
Jianxin, W., Jiayu, X. & Yaozong, Y. Effects and mechanisms of emodin and sandostatin on pancreatic ischemia in acute haemorrhagic necrotizing pancreatitis., CJIM 4, 34–38 (1998). https://doi.org/10.1007/BF02935047DOI:
Wu Jianxin, Xu Jiayu, Yuan Yaozong. Effects and mechanisms of emodin and sandostatin on pancreatic ischemia in acute haemorrhagic necrotizing pancreatitis. [J]. Chinese Journal of Integrative Medicine 4(1):34-38(1998) DOI: 10.1007/BF02935047.
Effects and mechanisms of emodin and sandostatin on pancreatic ischemia in acute haemorrhagic necrotizing pancreatitis
摘要
Objective: To investigate pancreatic ischemia and abnormal metabolism of eicosanoids in acute haemorrhagic necrotizing pancreatitis (AHNP) and the effects of emodin or sandostatin on them. Methods: In rats with AHNP triggered with sodium taurocholate
the pancreatic blood flow (PBF) was detected with computerized tissue blood flowmeter
and plasma thromboxane B2(TXB2)
6-keto-prostaglandin Fla(6-keto-PGF1a) and prostaglandin E2(PGE2) were determined with radioimmunoassay. Results: There was a significant decrease of PBF in the early stage of AHNP. Compared with that in the non-treated group
significant improvement of PBF was demonstrated in emodin as well as in sandostatin group which showed reduced PBF following infusion of sandostatin before AHNP was triggered. In the non-treated group
plasma TXB2 was significantly higher
with an increase of 4.5 times
than that in sham-operated group
while 6-keto-PGF1a and PGE2 tended to decrease. The above mentioned abnormal metabolism of eicosanoids was blocked either in emodin or in sandostatin group in which lessened damage of acinar cells was shown by pathologic scoring or transmission electron microscope. Both groups shared significantly lower mortalities than the non-treated group. Conclusions: Either emodion or sandostatin could partly reverse the decrease of PBF in the early stage of AHNP
which may be ascribed at least in part to inhibition of abnormal metabolism of eicosanoids and improvement of pancreatic cytoprotection
and combined application of the two drugs is likely to have synergetic action.
Abstract
Objective: To investigate pancreatic ischemia and abnormal metabolism of eicosanoids in acute haemorrhagic necrotizing pancreatitis (AHNP) and the effects of emodin or sandostatin on them. Methods: In rats with AHNP triggered with sodium taurocholate
the pancreatic blood flow (PBF) was detected with computerized tissue blood flowmeter
and plasma thromboxane B2(TXB2)
6-keto-prostaglandin Fla(6-keto-PGF1a) and prostaglandin E2(PGE2) were determined with radioimmunoassay. Results: There was a significant decrease of PBF in the early stage of AHNP. Compared with that in the non-treated group
significant improvement of PBF was demonstrated in emodin as well as in sandostatin group which showed reduced PBF following infusion of sandostatin before AHNP was triggered. In the non-treated group
plasma TXB2 was significantly higher
with an increase of 4.5 times
than that in sham-operated group
while 6-keto-PGF1a and PGE2 tended to decrease. The above mentioned abnormal metabolism of eicosanoids was blocked either in emodin or in sandostatin group in which lessened damage of acinar cells was shown by pathologic scoring or transmission electron microscope. Both groups shared significantly lower mortalities than the non-treated group. Conclusions: Either emodion or sandostatin could partly reverse the decrease of PBF in the early stage of AHNP
which may be ascribed at least in part to inhibition of abnormal metabolism of eicosanoids and improvement of pancreatic cytoprotection
and combined application of the two drugs is likely to have synergetic action.
关键词
PancreatitisischemiaEmodinsandostatineicosanoids
Keywords
PancreatitisischemiaEmodinsandostatineicosanoids
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